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2015). These scientific tests, taken with each other, suggest that, Though a immediate activation of GPR55 or PPAR‐α occurs, PEA can generate its anti‐inflammatory motion inside the gut also by means of oblique activation of CB1 and CB2 receptors, possibly due to the ability of this compound to potentiate the action of endocannabinoids at thes
In these problems, it has been shown that the increase of endogenous Palmitoylethanolamide—both by decreasing its degradation or exogenous administration—is ready to retain neuroinflammation inside of its physiological limitations. With this critique the large variety of experiments on the benefits derived from oral administration of micronized